Robert David STEELE Vivas
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Hillary Clinton Room, Women?s National Democratic Club
Who?s Who In Collective Intelligence: Robert David STEELE Vivas
Sense of Humor Below the Line
Robert David STEELE Vivas
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Nov 19
Source: http://www.phibetaiota.net/2012/11/reference-robert-david-steele-vivas-at-60/
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Fort Campbell, KY ? Don?t let the cold weather keep you indoors.? Outdoor Recreation Adventure Programs has just the thing to help you beat that holiday stress. They start the month of December with a Chickasaw Trace Mountain Biking trip on December 1st from 8:00am until 4:00pm. Ages 14 and above are invited to participate and the cost is $19.00.
Helmets, bikes and transportation are provided.? Chickasaw Trace is a small 300 acre park bordering the Duck River just southwest of Nashville, TN.? It may be small but this park offers some of the best mountain biking in Middle Tennessee. It has something to offer cyclists of all levels of experience.? Preregistration is required by 4:00pm on November 29th.
On December 8th they will be holding a Toy Drive at the Challenge Course from 1:00pm until 3:00pm. Don?t miss this great opportunity to help other have a great Christmas.? Just bring a toy for donation and Outdoor Recreation will waive your entrance fee to the Challenge Course. Toys will be donated to Operation Homefront.Visit the Golden Pond Planetarium for a meteor shower and Land Between the Lakes Planetarium holiday laser show on December 15th. The trip is scheduled for 4:00pm until 10:00pm and the cost is $13.00. Don?t forget to dress warm for this event!? Preregistration is required by 4:00pm on December 13th.
Enjoy a Savage Gulf Day Hike on December 16th from 8:00am until 6:00pm. Ages 12 and over may participate. The cost is $19.00 and includes transportation. Located on top of the Cumberland Plateau, the 15,000 acre Savage Gulf State Natural Area is one of Tennessee?s most pristine wilderness areas. With over 50 miles of hiking trails, Savage Gulf offers hikers some of the most stunning views in the state. Bring your camera and your sense of adventure because this is one trip you don?t want to miss! Preregistration is required by 4:00pm on December 14th.
Whether your pooches are big or small, they will love spending time with their owners and making new puppy pals while on a Dog Hike. You?ll be hiking the trails located around Outdoor Recreation on December 29th from 10:00am until 12:00pm. Dogs must remain on a leash at all times. This cost is $5.00 and preregistration is required by 4:00pm on December 28th.
For more information about their December activities, please call Outdoor Recreation Adventure Programs at 270.412.7854.
Source: http://www.clarksvilleonline.com/2012/11/20/fort-campbells-outdoor-recreation-december-activities/
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Writing in the journal Science, Professor Derby of The School of Materials, looks at how the concept of using printer technology to build structures in which to grow cells, is helping to regenerate tissue.
Both inkjet and laser printer technology can be used to build the 3D scaffolds that cells can be grown in and also place the cells in these structures simultaneously. Professor Derby explains how bioprinting works: "Inkjet technology places the structure's material in small droplets, which then solidify. More droplets are then placed on top of the previous ones in a specific pattern. The structure is built using this method which is generally referred to as additive manufacture.
"Laser printing uses light to solidify the structure's material layer upon layer. These methods have allowed us to develop very complex scaffolds which better mimic the conditions inside the body."
The scaffold provides a surface for the cells to adhere, thrive and multiply. Both the scaffold material, composition and its internal architecture control the behaviour and well-being of the cells inside.
In his review article Professor Derby looks at experiments where porous structures have been made through bioprinting. They are then placed in the body to help act as a scaffold to encourage cell growth. The cells colonize the structure and it either dissolves or becomes part of the body.
This type of treatment can help patients suffering from problems such as cavity wounds. Clinical trials are currently taking place around the world to perfect this technology, and Professor Derby says it is moving towards becoming an established form of science.
Professor Derby also looks at how stem cells are being grown in printed structures that have been impregnated with certain chemicals. The chemicals are inserted during the printing process and can determine the type of cell the stem cells develop into. For example stem cells could be programmed to become cells that make up bone tissue or cartilage.
But there are limitations to the technology which is holding back breakthroughs such as the ability to grow an entire organ. Studies have found that it is very difficult to actually print the cells at the same time as making the structure that will house them. The stress on the cell as it goes through both the inkjet and laser process can damage the cell membrane. Cell survival rates have also been variable, ranging from between 40 to 95%.
The technology is also some way off progressing from an experimental platform to clinical practice. Whilst scaffolds are being clinically trialled, actually transplanting cells grown in an external structure into a patient is a more advanced process. It is still not possible at present to guarantee a consistent quality, which is required by medical device regulations.
But research is being carried out to grow external cells into tissue, such as a patch of skin, and transplant that into a patient. Professor Derby is currently working with Ear, Nose and Throat surgeons at the Manchester Royal Infirmary. He wants to use bioprinting to print cells without using a scaffold. The printed cells form a sheet that can be used for grafts inside the body, for example in the mouth or nose.
Professor Derby says: "It is very difficult to transplant even a small patch of tissue to repair the inside of the nose or mouth. Current practice, to transplant the patient's skin to these areas, is regarded as unsatisfactory because the transplants do not possess mucous generating cells or salivary glands. We are working on techniques to print sheets of cells that are suitable for implantation in the mouth and nose."
One area which Professor Derby's review article highlights for the future is the ability to grow structures which can model cancerous tumours. These could then be used to test new drugs, which it's hoped will advance the search for more effective treatments.
Writing the review article has encouraged Professor Derby that there is a strong future for bioprinting and whilst growing organs is still a long way off, the advances being made in this area are very promising.
###
University of Manchester: http://www.manchester.ac.uk
Thanks to University of Manchester for this article.
This press release was posted to serve as a topic for discussion. Please comment below. We try our best to only post press releases that are associated with peer reviewed scientific literature. Critical discussions of the research are appreciated. If you need help finding a link to the original article, please contact us on twitter or via e-mail.
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Source: http://www.labspaces.net/125314/Bioprinting_has_promising_future
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News outlets reported on the ongoing public and behind-the-scenes discussions about the fiscal cliff, including changes to Medicare.
The Wall Street Journal: Pelosi: Too Much at Risk to Go Over The Cliff
House Minority Leader Nancy Pelosi (D., Calif.) said Sunday she wants to complete a deal to avoid a year-end fiscal crisis next month, brushing aside calls from the liberal wing of the Democratic caucus who say the country might be better off if Congress did nothing. ... Democratic and Republican leaders emerged from a meeting on Friday uncharacteristically optimistic they would find an agreement. Aides said the leaders agreed to a two-step approach that would make a down payment on deficit reduction this year, forcing decisions next year on bigger issues such as overhauling the tax code and making changes to entitlement programs such as Medicare (Hughes and Johnson, 11/18).
The New York Times: At Bipartisan Budget Meeting, Familiar Hurdles But A New Attitude?
As tentatively envisioned, a compromise would provide an immediate down payment of at least $50 billion to reduce this year's projected deficit ... Second, it would define a framework for negotiating a long-term "grand bargain" in 2013 to shave annual deficits by perhaps $4 trillion over the first decade. The framework would have separate goals for raising revenues and cutting the two types of federal spending: so-called discretionary financing that Congress sets annually for most programs, domestic and military; and entitlement spending, chiefly for Medicare and Medicaid, which by their growth in an aging population are driving projections of mounting debt (Calmes and Weisman, 11/16).
The Washington Post: Both Sides Appear Upbeat On Opening Round Of 'Fiscal Cliff' Talks?
Since Nov. 6, when Obama won reelection and Democrats made gains in the House and Senate on a pledge to make the rich "pay their fair share," Republicans have backed down from the fight. On Friday, (Senate Minority Leader Mitch) McConnell and (House Speaker John) Boehner acknowledged the need for fresh revenue to restrain a public debt that has swollen to dangerous levels ? as long as Democrats agree to tackle the rising cost of Medicare and Medicaid, the biggest drivers of future borrowing (Montgomery and Goldfarb, 11/16).
The Hill: Ayotte: No Higher Tax Rates In 'Fiscal Cliff' Deal
A deal to avoid the "fiscal cliff" should include entitlement reforms as well as new revenues ? but not higher tax rates ? Sen. Kelly Ayotte (R-N.H.) said in the GOP's weekly address. Republicans have warmed to the idea of new revenues since President Obama's reelection, but lawmakers are still divided over whether to raise tax rates on the wealthy or find money elsewhere (Baker, 11/17).
The Hill: Obama: Only Two Paths Available On 'Fiscal Cliff'
During his weekly address on Saturday, President Obama warned Republicans that there are only two possible outcomes for negotiations over the looming "fiscal cliff." He said that either they agree to allow tax breaks to expire for the top two percent of American earners, or taxes will go up for everyone (Sasso, 11/17).
Los Angeles Times: Obama, Sen. Ayotte Talk 'Fiscal Cliff' In Weekly Addresses
Even as both parties signaled they were making progress on a deal to avoid the so-called fiscal cliff, President Obama on Saturday renewed his call on the House of Representatives to act in the short term to extend lower tax rates for the bottom tier of wage earners, saying it would boost Americans? confidence ahead of the holidays (Memoli, 11/16).
The Associated Press: Democrats Toughen Stance On Trimming Benefits
President Barack Obama's re-election has stiffened Democrats' spine against cutting popular benefit programs such as Medicare and Social Security. Their new resolve could become as big a hurdle to a deal that would skirt crippling tax increases and spending cuts in January as Republicans' resistance to raising tax rates on the wealthy.?Just last year, Obama and top Democrats were willing during budget negotiations with Republicans to take politically risky steps such as reducing the annual inflation adjustment to Social Security and raising the eligibility age for Medicare (Taylor, 11/17).
The Washington Post: AARP Uses Its Power To Oppose Social Security, Medicare Benefit Cuts For Retirees
AARP?s rejection of any significant changes to the nation?s safety net could be a major factor as policymakers seek a deal to put the government?s finances in order through raising taxes and cutting spending on federal programs, possibly including popular entitlements such as Medicare and Social Security. ... AARP opposes raising the age for Medicare eligibility on the grounds that it would increase costs for younger seniors while driving up premium costs for older ones (Fletcher and Goldfarb, 11/17).
This is part of Kaiser Health News' Daily Report - a summary of health policy coverage from more than 300 news organizations. The full summary of the day's news can be found here and you can sign up for e-mail subscriptions to the Daily Report here. In addition, our staff of reporters and correspondents file original stories each day, which you can find on our home page.
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Public release date: 19-Nov-2012
Share ] Contact: presse
presse@inserm.fr
INSERM (Institut national de la sant et de la recherche mdicale)
A large-scale international study involving French researchers from the Inserm-Institut Pasteur Lille-Universit Lille Nord de France "Public health and molecular epidemiology of ageing-related diseases" joint research unit led by Philippe Amouyel, has just discovered a gene for susceptibility to a rare disease that causes susceptibility to a common one, Alzheimer's disease, providing evidence of the heterogeneous aetiology of Alzheimer's disease.
This whole-exome sequencing approach is explained in detail in The New England Journal of Medicine dated 14 November 2012.
Polycystic lipomembranous osteodysplasia with sclerosing leukoencephalopathy, or Nasu-Hakola disease, is a genetic disorder passed on by means of autosomal recessive transmission. The disease starts at the age of around 30 years with pains in the wrists or shoulders associated with swollen joints. Bone fractures can occur as a result of minor traumas. Bone x-rays show epiphyseal cysts. Slight personality changes then occur followed by frontal neurological signs (euphoria, loss of social inhibitions) evolving into early-onset dementia. The disorder has been associated with mutations of the TREM2 (Triggering Receptor Expressed on Myeloid cells 2) gene on chromosome 6.
British, American and French researchers have now shown that on this same region of chromosome 6, mutations of the TREM2 gene were associated with a five times greater risk of developing late-onset Alzheimer's disease. A complete sequencing was performed on 281 individuals with Alzheimer's disease and 504 controls. Analysis of the TREM2 gene showed excessive TREM2 mutations in those with the disease compared with the control subjects. Characterisation of one of these TREM2 mutations in very large sample populations of patients with Alzheimer's disease has allowed researchers to measure precisely the importance of this association between TREM2 mutations and the disease. Finally, a replication study was performed in another independent series of 1994 cases and 4602 controls, which confirmed this strong association (OR=4.97 CI 95% [2.42-10.21], P
These results are also confirmed in the same edition of The New England Journal of Medicine by an Icelandic team, which also shows that this gene is a risk factor for Alzheimer's disease in the Finnish population and other European populations.
A pathological analysis of six individuals presenting variants of the TREM2 gene has revealed evidence of Alzheimer's-type brain lesions. The study of TREM2 gene expression in normal human brains has shown high levels in the white matter and in the hippocampus and cortex.
In a transgenic mouse model of Alzheimer's disease, an increase in TREM2 expression was observed in microglial cells surrounding the amyloid plaques and the neurons compared with normal mice. The TREM2 gene encodes a protein that participates in the activation of immune responses in macrophages and dendritic cells.
This discovery has two main consequences. Firstly, this observation provides a better understanding of the immune system's involvement in Alzheimer's disease in which the gene of complement receptor 1 (CR1) had already been implicated, in previous work by Inserm-Lille2-IPL UMR744 . Furthermore, this approach of whole-exome sequencing has allowed the discovery of a gene for susceptibility to a rare disease that causes susceptibility to a common disease, evidence of the heterogeneous aetiology of Alzheimer's disease. It is the loss of function of this gene in its homozygous or heterozygous variants that determines the nature of the disorder.
These results, which demonstrate how much progress has been made in understanding Alzheimer's disease, involved teams from LabEx DISTALZ, and were able to be produced partly through the support of the French Foundation for Scientific Cooperation on Alzheimer's and similar diseases, launched in February 2008.
###
Ever-increasing longevity among the human population means the number of patients suffering from Alzheimer's disease is also on the rise in France and throughout the world. Alzheimer's is the leading cause of memory and intellectual function disorders among elderly people and represents a major public health issue.
Alzheimer's disease is one of the main causes of dependency among the elderly. It results from neurodegeneration in different areas of the brain. Its symptoms include increasing impairment of memory and cognitive functions, and behaviour disorders that lead to a progressive loss of independence. In France, Alzheimer's disease affects more than 850,000 people and represents major social and economic costs.
Alzheimer's disease is characterized by the development of two types of lesion in the brain: amyloid plaques and neurofibrillary tangles. Amyloid plaques originate from the extracellular accumulation of a peptide, the beta amyloid peptide, in specific areas of the brain. Neurofibrillary tangles are intraneuronal lesions caused by abnormal filamentary aggregation of a protein known as a tau protein.
Identifying the genes that participate in the incidence of Alzheimer's disease and its development will make it possible to tackle the physiopathological mechanisms behind this affliction more rapidly, to identify the target proteins and metabolic channels for new treatments, and to provide a means of identifying the individuals that are most at risk when effective preventive treatments become available.
[ | E-mail | Share ] ?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Public release date: 19-Nov-2012 Share ] Contact: presse
presse@inserm.fr
INSERM (Institut national de la sant et de la recherche mdicale)
A large-scale international study involving French researchers from the Inserm-Institut Pasteur Lille-Universit Lille Nord de France "Public health and molecular epidemiology of ageing-related diseases" joint research unit led by Philippe Amouyel, has just discovered a gene for susceptibility to a rare disease that causes susceptibility to a common one, Alzheimer's disease, providing evidence of the heterogeneous aetiology of Alzheimer's disease.
This whole-exome sequencing approach is explained in detail in The New England Journal of Medicine dated 14 November 2012.
Polycystic lipomembranous osteodysplasia with sclerosing leukoencephalopathy, or Nasu-Hakola disease, is a genetic disorder passed on by means of autosomal recessive transmission. The disease starts at the age of around 30 years with pains in the wrists or shoulders associated with swollen joints. Bone fractures can occur as a result of minor traumas. Bone x-rays show epiphyseal cysts. Slight personality changes then occur followed by frontal neurological signs (euphoria, loss of social inhibitions) evolving into early-onset dementia. The disorder has been associated with mutations of the TREM2 (Triggering Receptor Expressed on Myeloid cells 2) gene on chromosome 6.
British, American and French researchers have now shown that on this same region of chromosome 6, mutations of the TREM2 gene were associated with a five times greater risk of developing late-onset Alzheimer's disease. A complete sequencing was performed on 281 individuals with Alzheimer's disease and 504 controls. Analysis of the TREM2 gene showed excessive TREM2 mutations in those with the disease compared with the control subjects. Characterisation of one of these TREM2 mutations in very large sample populations of patients with Alzheimer's disease has allowed researchers to measure precisely the importance of this association between TREM2 mutations and the disease. Finally, a replication study was performed in another independent series of 1994 cases and 4602 controls, which confirmed this strong association (OR=4.97 CI 95% [2.42-10.21], P
These results are also confirmed in the same edition of The New England Journal of Medicine by an Icelandic team, which also shows that this gene is a risk factor for Alzheimer's disease in the Finnish population and other European populations.
A pathological analysis of six individuals presenting variants of the TREM2 gene has revealed evidence of Alzheimer's-type brain lesions. The study of TREM2 gene expression in normal human brains has shown high levels in the white matter and in the hippocampus and cortex.
In a transgenic mouse model of Alzheimer's disease, an increase in TREM2 expression was observed in microglial cells surrounding the amyloid plaques and the neurons compared with normal mice. The TREM2 gene encodes a protein that participates in the activation of immune responses in macrophages and dendritic cells.
This discovery has two main consequences. Firstly, this observation provides a better understanding of the immune system's involvement in Alzheimer's disease in which the gene of complement receptor 1 (CR1) had already been implicated, in previous work by Inserm-Lille2-IPL UMR744 . Furthermore, this approach of whole-exome sequencing has allowed the discovery of a gene for susceptibility to a rare disease that causes susceptibility to a common disease, evidence of the heterogeneous aetiology of Alzheimer's disease. It is the loss of function of this gene in its homozygous or heterozygous variants that determines the nature of the disorder.
These results, which demonstrate how much progress has been made in understanding Alzheimer's disease, involved teams from LabEx DISTALZ, and were able to be produced partly through the support of the French Foundation for Scientific Cooperation on Alzheimer's and similar diseases, launched in February 2008.
###
Ever-increasing longevity among the human population means the number of patients suffering from Alzheimer's disease is also on the rise in France and throughout the world. Alzheimer's is the leading cause of memory and intellectual function disorders among elderly people and represents a major public health issue.
Alzheimer's disease is one of the main causes of dependency among the elderly. It results from neurodegeneration in different areas of the brain. Its symptoms include increasing impairment of memory and cognitive functions, and behaviour disorders that lead to a progressive loss of independence. In France, Alzheimer's disease affects more than 850,000 people and represents major social and economic costs.
Alzheimer's disease is characterized by the development of two types of lesion in the brain: amyloid plaques and neurofibrillary tangles. Amyloid plaques originate from the extracellular accumulation of a peptide, the beta amyloid peptide, in specific areas of the brain. Neurofibrillary tangles are intraneuronal lesions caused by abnormal filamentary aggregation of a protein known as a tau protein.
Identifying the genes that participate in the incidence of Alzheimer's disease and its development will make it possible to tackle the physiopathological mechanisms behind this affliction more rapidly, to identify the target proteins and metabolic channels for new treatments, and to provide a means of identifying the individuals that are most at risk when effective preventive treatments become available.
[ | E-mail | Share ] ?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Source: http://www.eurekalert.org/pub_releases/2012-11/ind-anf111912.php
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Distributing promotional SEO videos is now the trend among companies who desire to reach their target audience fast. In accordance to the purpose they have in mind, businesses create and post clips of different nature. They vary from tutorials to testimonials to free online courses.
Internet users desire to acquire information as quick as possible. In response to this, businesses are creating and posting relevant clips online in a snap. They do this without even thinking about the security of their original content. Owners or producers must understand that without proper security, their masterpieces can be easily distributed without their permission.
To combat similar situations from happening to your business, here are some insights which can help you secure all your promotional videos.
Select Online Streaming ? Online streaming is the option most owners choose today over progressive download. Progressive download is as effective as online streaming when it comes to information dissemination. However, it is online streaming which allows full control over social media content distribution.
The birth of YouTube brought about the existence of progressive download. As security is not tight with this hosting option, anyone can simply watch, download and save video files in their computer without asking the permission of their owners. Also, there is liberty to repurpose these files to their hearts content.
Most companies today make use of web video production to earn income online. They reach out to their target market by offering helpful and original content. This is especially true for those who do short courses, if not for free, come with minimal fees. With online streaming, you can limit the extent of the content you are to distribute. You do not have to worry about your content being copied as saving video files is not allowed at all.
Create Access Controls ? Who can access your clips online? That is the question you need to answer even before the creation of your videos begin. Exclusive content has to be protected at all cost. Once it gets replicated without your permission all your months of research would be useless. Authorize specific people who can view your videos.
Secure Sockets Layer ? The Secure Sockets Layer allows the encryption of your video stream to come in between the server and the browser of the internet user. This process secures all important data about your video in the form of illegible and incomprehensible text in the internet. This is the opposite of what happens in YouTube. You would notice that, in there, everything about the video appear in readable characters. More so, the message they convey is understandable.
The lack of security has caused many of the excellent clips produced online illegally replicated and distributed with or without the knowledge of their makers. Perhaps, you did not know any measure to secure your promotional videos then. Now that you already have the idea, take the effort to do so for a well-meaning distribution of your original content.
Looking For Tips On Internet Viral Marketing? Let Video Marketing Expert David Jenyns Help Produce Your Viral Videos.
Source: http://wedonetwork.co.uk/wedotech/2012/11/19/top-tips-in-protecting-your-promotional-videos/
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IMAGINE yourself at a magic show. The magician brings out a tiger and coaxes it into a large, colorful box on the stage. He closes the lid, says a few mysterious words and then ? poof ? opens the side panel, revealing the inside of the box to be empty. The tiger is gone. Cue applause.
We know, of course, that tigers are not apt to vanish into thin air; we know that such magic tricks are more trick than magic. But how is it possible that our eyes can be deceived so easily?
The answer has much to do with the way our sense of sight works. As we look around a room, our eyes detect the light that bounces off nearby people or objects, and our brains interpret the images formed from the patterns of light received. We can even figure out what material something is made of based on the way it reflects and transmits light: metal is opaque and typically very reflective; plastic, which is more dull and often translucent, absorbs some of the light and reflects the rest in all directions. Our brains, then, turn these signals from reflections into breathtakingly complex pictures of the world around us. And it all happens faster than the blink of an eye. Indeed, after every blink of an eye.
Such lightning-fast cognitions are possible partly because the brain makes certain automatic assumptions: it figures that light has traveled in a straight line from the object to our eyes. Remarkably, in that built-in assumption is the recipe for a bit of magic that humans (and mythical humans) have sought, from the time of Plato to the age of Harry Potter: invisibility.
The trick involves the ability to bend and distort light as it travels through space ? in other words, to make it do what the brain assumes it won?t. In some ways, it?s the same sleight of hand that the magician uses with the tiger. He uses a mirror angled in such a way that when we think we?re looking into an empty box, we?re actually seeing the reflection from the bottom of the box and assuming it?s the back. Since we don?t expect that the light reaching our eyes has swerved, making a 90-degree turn along the way, our eyes ?tell? us the tiger has vanished. (In reality, he?s hiding comfortably in the box.)
Now we?ve found a way to one-up this neat trick with science: changing the trajectory of light without using mirrors. We do it with the science of materials ? designing a ?cloak? that can make light curve around an object, and then emerge just as if it had passed in a straight line through space. (Think of it like water flowing past a rock in a stream.)
The phenomenon is indeed supernatural. That?s because nature doesn?t appear to offer any materials that can accomplish this feat. The reason is that light has both electric and magnetic components ? and to make it swerve around an object, one has to redirect both of these very different components and have them sync up immediately after the detour. That?s impossible to do with metals, fabrics or any other traditional materials.
But research findings over the past decade have shown us how to develop artificially structured ?metamaterials? ? in which tiny electrical circuits serve as the building blocks in much the same way that atoms and molecules provide the structure of natural substances. By changing the geometry and other parameters of those circuits, we can give these materials properties beyond what nature offers, letting us simultaneously manipulate both the electric and magnetic aspects of light in striking harmony.
This year, with one such metamaterial, we built the world?s first invisibility cloak capable of managing both components of light.
There is a catch, admittedly. Our cloak works only on microwaves, not on visible light. And humans don?t ?see? microwaves in the first place, making the idea of invisibility seem, well, a little extraneous.
Still, even if we mortals don?t see them, many essential devices do. Nearly every time you walk through security at an airport, your body is scanned with microwaves. Also, your cellphone, iPad and other devices make a similar kind of virtual eye contact with one another. So, even in the microwave realm, cloaking can potentially be used to remove obstacles from the paths of direct microwave communications (or hide things we don?t want detected).
More important, microwaves are part of the same electromagnetic spectrum as visible light. In principle, if cloaks can be made to work at microwave frequencies, they might one day be made to work at visible wavelengths.
This will be far more difficult: the wavelengths of visible light are more than 10,000 times smaller than those of microwaves, meaning that the corresponding metamaterials would have to be equally reduced in size.
What excites scientists and Harry Potter fans alike, though, is that our microwave cloak proves there?s no theoretical limitation that would prevent someone from building a visible-light cloak.
There are some tricky technological barriers to work out. But in this case, at least, not seeing is believing.
David R. Smith is a professor of electrical and computer engineering at Duke University, where Nathan Landy is a graduate student.
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Is it just me, or does Ron have almost the exact same singing voice as Rodney Anonymous from the Dead Milkmen?
Looks like one kick ass wedding! And this is coming from a guy who?s wedding was basically the ?short short version? from Spaceballs.
Big congrats to Darwyn Cooke and his new bride and great job Ron!
Source: http://feedproxy.google.com/~r/ifanboy/~3/kQA6jgdvwMc/
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Nairobi ? The central Organisation of Trade Unions is now demanding the registration of a police union to enable those who enforce the law in Kenya air their grievances and push for better working conditions.
Speaking on Sunday, Secretary general Francis Atwoli said COTU wants the union registered within seven days.
He argued that trade unions for police officers exist in many countries across the world and it was not an outlandish proposition.
"We have police unions in countries like South Africa which even has two of them, Norway is the oldest... in Sweden we have the same. We have one in Australia, USA and many others. The key thing is to have a rider that ensures that they must always engage in dialogue and strike be the last option," he added.
"These are our daughters and sons who were ambushed and killed while trying to protect poor Kenyans. They must have a document that spells out their terms and conditions of service, ranging from housing, insurance cover, leave, wages, overtime that they perform, education of their children and their spouses, and provision of health care," Atwoli said.
He said such a union would open a channel for dialogue between the government and police officers and not encourage strikes.
"Those unions that are under COTU with their full Collective Bargaining Agreements (CBA), how many of them go on strike? Almost nil. Because they are always in boardrooms negotiating as per the requirement of the CBA. So if the police are allowed to enter into a dialogue on issues of importance with the government, they cannot go on strike," he argued.
Currently picketing is outlawed in the constitution for members of the disciplined forces.
Atwoli also expressed his disappointment at the manner in which the government through the minister of Internal Security has handled the issue of the death of the 48 police officers who were killed in Baragoi, Samburu while in a mission to flush out cattle rustlers.
He said the matter has been handled randomly.
"It's a pity up to now... you can read all the newspapers that even issues to do with compensation and what have you, no one is talking! the burial programme; no one is talking about it. And it is simply because, our officers in uniform are not effectively represented by a trade union," Atwoli lamented.
The demand by COTU comes amid concerns police officers work under inhumane conditions and are ill-equipped to perform their duties leading to deaths in the line of duty.
COTU now plans to have a special committee to follow up the matter and ensure a police union is registered in a week's time.
"We will call you for the next action, if the government does not act," Atwoli vowed.
Source: http://allafrica.com/stories/201211180540.html
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Galapagos National Park via AP
Galapagos National Park staff on Monday test equipment that will hold poisonous bait to kill rats.
By Gonzalo Solano, Associated Press
QUITO, Ecuador -- The unique bird and reptile species that make the Galapagos Islands a treasure for scientists and tourists must be preserved, Ecuadorean authorities say ? and that means the rats must die, starting with 180 million of them.
?
A helicopter on Thursday began dropping specially designed poison bait on an island Thursday, launching the second phase of a campaign to clear out by 2020 non-native rodents from the archipelago that helped inspire Charles Darwin's theory of evolution.
The invasive Norway and black rats, introduced by whalers and buccaneers beginning in the 17th century, feed on the eggs and hatchlings of the islands' native species, which include giant tortoises, lava lizards, snakes, hawks and iguanas. Rats also have depleted plants on which native species feed.
The rats have critically endangered bird species on the 19-island cluster 600 miles from Ecuador's coast.
"It's one of the worst problems the Galapagos have. (Rats) reproduce every three months and eat everything," said Juan Carlos Gonzalez, a specialist with the Nature Conservancy. While his organization has no involvement in the Phase II eradication operation, on Pinzon island and the islet of Plaza Sur, he said it has no objections.
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Phase I of the anti-rat campaign began in January 2011 on Rabida island and about a dozen islets, which like Pinzon and Plaza Sur are also uninhabited by humans.
The goal is to kill off all nonnative rodents, beginning with the Galapagos' smaller islands, without endangering other wildlife. The islands where humans reside, Isabela and Santa Cruz, will come last.
Previous efforts to eradicate invasive species have removed goats, cats, burros and pigs from various islands.
Pinzon is about seven square miles in area, while Plaza Sur encompasses just 24 acres.
"This is a very expensive but totally necessary war," said Gonzalez.
The rat infestation has now reached one per square foot on Pinzon, where an estimated 180 million rodents reside.
The director of conservation for the Galapagos National Park Service, Danny Rueda, called the raticide the largest ever in South America.
Galapagos' Lonesome George might not have been last of his kind
The poisoned bait, developed by Bell Laboratories in the United States, is contained in light blue cubes that attract rats but are repulsive to other inhabitants of the islands. The one-centimeter-square cubes disintegrate in a week or so.
Park official Cristian Sevilla said the poison will be dropped on Pinzon and Plaza Sur through the end of November.
A total of 34 hawks from Pinzon were trapped in order to protect them from eating rodents that consume the poison, Sevilla said. They are to be released in early January.
On Plaza Sur, 40 iguanas were also captured temporarily for their own protection.
Asked whether a large number of decomposing rats would create an environmental problem, Rueda said the poison was specially engineered with a strong anti-coagulant that will make the rats dry up and disintegrate in less than eight days without a stench.
It will help that the average temperature of the islands is 75 degrees Fahrenheit, he added.
The current $1.8 million phase of the project is financed by the national park and nonprofit conservation groups including Island Conservation.
The Galapagos were declared protected as a UNESCO Natural Heritage site in 1978. In 2007, UNESCO declared them at risk due to harm from invasive species, tourism and immigration.?
More world stories from NBC News:
Follow World News from NBCNews.com on Twitter and Facebook
? 2012 The Associated Press. All rights reserved. This material may not be published, broadcast, rewritten or redistributed.
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As long as organized sports have captivated the general public, there have been head-scratching rules governing play and conduct that make zero sense. Up until the start of the 2012-13 college basketball season, the sometimes-arcane rules of golf took the prize for stupidity, but now, the leaders in the clubhouse have been challenged.
Not by the NCAA, the NFL, or Major League Baseball, but by the athletic department at the University of Washington.
Social media is sometimes a polarizing topic, but usually the issues revolve around student-athletes using Facebook and Twitter irresponsibly. Not so in Nirvana country, though, as apparently even the most rogue student-athlete doesn?t hold a candle to the real menaces to society: Sportswriters.
The University of Washington has instituted a policy where credentialed reporters at UW home basketball games cannot tweet more than 20 times from beginning to end. In fact, assorted higher-ups in the athletic department actually reprimanded Todd Dybas of the Tacoma News Tribune for excessive tweeting during the Huskies? season-opening win over Loyola (Md.) earlier this week.
My initial response to this was out-and-out laughter at the institution. I understand wanting to control a message, especially during a time of crisis. But does this mean someone, somewhere, was actually getting paid to count tweets by reporters at the game? Furthermore, how long until the university revokes a reporter?s pass for simply doing his or her job?
A look around the PAC-12 reveals that conference mate USC has a similar policy in place. What sense does this make? If reporters want to cover a team?s games and/or practices, Twitter is a valuable tool to keep fans informed when news breaks. It produces excitement, keeps a program in the news and raises awareness of upcoming games and promotions.
Why on Earth would UW and USC restrict tweets from reporters?
?I think just generally speaking is what we?re trying to do is steer people toward partnerships we have with radio, television and our own web presence,? USC director of social media Jordan Moore said in an interview with website GeekWire earlier this week. ?We don?t want people taken away from that experience.?
So the athletic departments want to restrict the flow of information that?s already readily accessible? There?s no reason to control a particular message when a crisis does not exist.
?We?re always going to protect live descriptions of events,? UW athletic director Scott Woodward said on Dave ?Softy? Mahler?s Seattle-area radio show Tuesday morning. ?That?s something that has always been our right for decades and continues to be so. As technology gets better and better, we?re going to have to be more vigilant about how we do it, but also understand that reporting has changed, too. There?s a fine line there and we?re always going to be cognizant and reasonable.? Continued...
So it?s reasonable to yell at Todd Dybas for doing his job? Would he prefer that Dybas and reporters like him follow Seattle coach Cameron Dollar?s advice and go to Seattle University games, where Dollar says they?d be welcomed with open arms?
None of this makes any rational sense. As a sportswriter, if I see something newsworthy happen at a sporting event I?m covering, you can bet your bottom dollar it?s going to be tweeted. As a fan, if I see something big happen at a game I?m attending or watching on TV, and a trusted reporter says or tweets nothing about it, I tend to lose a bit of faith in that source.
I?d like to think that those in my profession feel the same way. Judging by the outcry spawned by the Twitter policy, it looks that way. One reporter, in covering Washington?s stunning loss to UAlbany, tweeted that the school would probably lose, then expand its media policy so nobody could report it. As outrageous as it may seem, would that really be a shock considering the athletic department?s recent behavior?
It?s my sincere hope that the school revisits and repeals the policy, one that benefits nobody and hinders reporters from doing their jobs. There are more important things to worry about than controlling problems that don?t exist. With some luck, common sense will prevail, not just at Washington and USC, but at all college athletic departments around the country.
Andrew Champagne is a sportswriter at the Saratogian. He can be reached at @AndrewChampagne on Twitter and via email at achampagne@saratogian.com.
Source: http://saratogian.com/articles/2012/11/15/sports/doc50a5a33b9cdf0040774657.txt
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MIDLAND, Texas (Reuters) - All four men killed when a freight train collided with a parade float honoring wounded American war veterans had served in the U.S. Army, police said on Friday, as investigators descended on the West Texas oil town of Midland to search the wreckage.
At least 16 other people were injured during the parade on Thursday at the start of what was meant to be a weekend of events including a banquet and a hunting expedition to salute U.S. veterans of the Iraq and Afghanistan wars.
Midland police identified the dead as: Sergeant Major Gary Stouffer, 37; Sergeant Major Lawrence Boivin, 47; Sergeant Major William Lubbers, 43; and Sergeant Joshua Michael, 34.
In Washington, the Army said Lubbers was retired from the military but could not immediately confirm whether the others were retired or active duty at the time of the accident.
The National Transportation Safety Board (NTSB) sent 16 experts to the scene to examine evidence including video from a forward-facing recorder on the train, officials said.
One person remained in critical condition and four were stable on Friday at a Midland hospital, a spokesman said. Another person was in serious but stable condition at a Lubbock hospital. Ten more were treated and released, police said.
The tragedy occurred as two flatbed trailers carrying the veterans, some of whom suffered major injuries in combat in Iraq and Afghanistan, attempted to cross railroad tracks during the "Hunt for Heroes" parade.
"The first flatbed crossed the train tracks completely. The second did not make it across before being struck by the train," a police statement said.
Veterans and their spouses jumped off the trailers to escape the collision. There were 26 people on the float that was hit by the Union Pacific train including a dozen veterans, a dozen spouses and two escorts, officials said.
"It's hard to look at. It's a very tragic event, very unfortunate," Midland Police Chief Price Robinson said from the site of the accident on Thursday.
The train and trailer remained in place on Friday morning at the site of the crash.
The NTSB said it would not determine a probable cause of the accident or provide any analysis while its investigators were at the scene. It said it had not yet determined if the grade-crossing warning system was working at the time of the accident.
A Union Pacific Corp spokesman referred questions on the investigation to the NTSB.
A woman who was riding a float that crossed the tracks ahead of the one struck in the accident said on Friday she heard a train horn and then people started to leap from both trailers.
"As soon as we crossed the tracks, we heard the train," Pam Shoemaker said, adding that she saw a railroad crossing bar start to come down just before the crash.
Many of the veterans honored in the event had served multiple deployments in Iraq and Afghanistan, according to event organizers. Some were said to have been shot or wounded by bombs and suffered traumatic brain injuries or post-traumatic stress.
The Pentagon said in a statement that Defense Secretary Leon Panetta, who is traveling in Asia, "was deeply saddened by news of the tragic accident."
Dr. Sudip Bose, a veteran who had been a frontline physician in Iraq, said he volunteered to provide medical help and was summoned to the crash scene. He said he had to set aside his emotions to tend to the injured.
"It was chaotic at times, gruesome at times," said Bose, a doctor in Odessa, Texas, who said veterans were applying tourniquets and pressure to injuries.
Midland Mayor Wes Perry and pastors Patrick Payton and Roy Smith held a prayer vigil on Friday morning attended by more than 100 people at a park, including veterans and family.
(Additional reporting by Phil Stewart, Mary Slosson and Barbara Goldberg; Editing by Daniel Trotta, David Bailey and Will Dunham)
Source: http://news.yahoo.com/train-collides-veterans-parade-texas-least-two-dead-004754241.html
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Willie Park Jr., a two-time winner of the British Open who later went on to make an impact on the sport as a teacher, author, course designer and club designer, will be inducted posthumously into the World Golf Hall of Fame May 6, 2013 at the Induction Ceremony held at the St. Johns Country Convention Center.
Park, who enters the Hall through the Veterans Category, joins his father, Willie Park Sr. The Parks will be the second father and son in the Hall of Fame, along with "Old Tom" Morris and "Young Tom" Morris.
Park Jr., a native of Scotland, won the Open Championships in 1887 at Prestwick and 1889 at Musselburgh, in his home town.
Park Jr. compiled 12 top-10 finishes in golf's oldest major. He was four years old when his father won the Open -- at Prestwick.
He went on to become one of the early pioneers in club and course design, and wrote the first golf instructional book by a professional player, "The Art of Putting," published in 1920.
Park Jr., designed or modified more than 200 courses in Europe, the U.S. and Canada. His three most significant designs are Sunningdale, near London, the Maidstone Golf Club in New York, and Royal Quebec.
Mungo Park, the great-nephew of Willie Park Jr., who serves as the family historian said that his uncle broke ground in professional golf.
"[Park Jr.], possibly more than any other, marked the transition from the old-school caddy and player to the modern professional golfer and businessman," Mungo Park said in a statement through the World Golf Hall of Fame.
Jack Peter, chief operating officer of the Hall of Fame, said having both Parks in the Hall of Fame "is truly fitting."
"The Hall of Fame celebrates the rich history of the game and that story cannot be told without the tremendous contributions of the Park family," Peter said in a statement.
Park Jr., will join Fred Couples and Ken Venturi in being inducted next May. A fourth member of the Class of 2013 will be announced soon.
Park died in 1925 at the age of 61.
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Public release date: 15-Nov-2012 Share ] Contact: Megan Watzke
mwatzke@cfa.harvard.edu
617-496-7998
Chandra X-ray Center
These images of the planetary nebula Abell 30, (a.k.a. A30), show one of the clearest views ever obtained of a special phase of evolution for these objects. The inset image on the right is a close-up view of A30 showing X-ray data from NASA's Chandra X-ray Observatory in purple and Hubble Space Telescope (HST) data showing optical emission from oxygen ions in orange. On the left is a larger view showing optical and X-ray data from the Kitt Peak National Observatory and ESA's XMM-Newton, respectively. In this image the optical data show emission from oxygen (orange) and hydrogen (green and blue), and X-ray emission is colored purple.
A planetary nebula so called because it looks like a planet when viewed with a small telescope is formed in the late stage of the evolution of a sun-like star.
After having steadily produced energy for several billion years through the nuclear fusion of hydrogen into helium in its central region, or core, the star undergoes a series of energy crises related to the depletion of hydrogen and subsequent contraction of the core. These crises culminate in the star expanding a hundred-fold to become a red giant.
Eventually the outer envelope of the red giant is ejected and moves away from the star at a relatively sedate speed of less than 100,000 miles per hour. The star meanwhile is transformed from a cool giant into a hot, compact star that produces intense ultraviolet (UV) radiation and a fast wind of particles moving at about 6 million miles per hour. The interaction of the UV radiation and the fast wind with the ejected red giant envelope creates the planetary nebula, shown by the large spherical shell in the bigger image.
In rare cases, nuclear fusion reactions in the region surrounding the star's core heat the outer envelope of the star so much that it temporarily becomes a red giant again. The sequence of events - envelope ejection followed by a fast stellar wind - is repeated on a much faster scale than before, and a small-scale planetary nebula is created inside the original one. In a sense, the planetary nebula is reborn.
The large nebula seen in the larger image has an observed age of about 12,500 years and was formed by the initial interaction of the fast and slow winds. The cloverleaf pattern of knots seen in both images, correspond to the recently ejected material. These knots were produced much more recently, as they have an observed age of about 850 years, based on observations of their expansion using HST.
The diffuse X-ray emission seen in the larger image and in the region around the central source in the inset is caused by interactions between wind from the star and the knots of the ejected material. The knots are heated and eroded by this interaction, producing X-ray emission. The cause of the point-like X-ray emission from the central star is unknown.
Studies of A30 and other planetary nebulas help improve our understanding of the evolution of sun-like stars as they near the end of their lifetime. The X-ray emission reveals how the material lost by the stars at different evolutionary stages interact with each another. These observations of A30, located about 5500 light years away, provide a picture of the harsh environment that the solar system will evolve towards in several billion years, when the sun's strong stellar wind and energetic radiation will blast those planets that survived the previous, red giant phase of stellar evolution.
The structures seen in A30 originally inspired the idea of reborn planetary nebulas, and only three other examples of this phenomenon are known. A new study of A30, using the observatories mentioned above, has been reported by an international team of astronomers in the August 20th, 2012 issue of The Astrophysical Journal.
###
The first author of the paper reporting these results is Martn A. Guerrero of the Instituto de Astrofsica de Andaluca (IAA-CSIC) in Spain. The other authors are N. Ruiz, also from the IAA-CSIC, Spain; W.-R. Hamann, from the University of Potsdam, Germany; Y.-H. Chu, from the University of Illinois, Urbana, IL; H. Todt, from the University of Potsdam, Germany; D. Schnberner, from the Leibniz-Institut Fr Astrophysik in Potsdam, Germany; L. Oskinova, from the University of Potsdam, Germany; R. Gruendl, from the University of Illinois, Urbana, IL; M. Steffen, from the Leibniz-Institut Fr Astrophysik in Potsdam, Germany; W. Blair, from Johns Hopkins University in Baltimore, MD and J. Toal from the IAA-CSIC, Spain.
NASA's Marshall Space Flight Center in Huntsville, Ala., manages the Chandra program for NASA's Science Mission Directorate in Washington. The Smithsonian Astrophysical Observatory controls Chandra's science and flight operations from Cambridge, Mass.
[ | E-mail | Share ] ?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Public release date: 15-Nov-2012 Share ] Contact: Megan Watzke
mwatzke@cfa.harvard.edu
617-496-7998
Chandra X-ray Center
These images of the planetary nebula Abell 30, (a.k.a. A30), show one of the clearest views ever obtained of a special phase of evolution for these objects. The inset image on the right is a close-up view of A30 showing X-ray data from NASA's Chandra X-ray Observatory in purple and Hubble Space Telescope (HST) data showing optical emission from oxygen ions in orange. On the left is a larger view showing optical and X-ray data from the Kitt Peak National Observatory and ESA's XMM-Newton, respectively. In this image the optical data show emission from oxygen (orange) and hydrogen (green and blue), and X-ray emission is colored purple.
A planetary nebula so called because it looks like a planet when viewed with a small telescope is formed in the late stage of the evolution of a sun-like star.
After having steadily produced energy for several billion years through the nuclear fusion of hydrogen into helium in its central region, or core, the star undergoes a series of energy crises related to the depletion of hydrogen and subsequent contraction of the core. These crises culminate in the star expanding a hundred-fold to become a red giant.
Eventually the outer envelope of the red giant is ejected and moves away from the star at a relatively sedate speed of less than 100,000 miles per hour. The star meanwhile is transformed from a cool giant into a hot, compact star that produces intense ultraviolet (UV) radiation and a fast wind of particles moving at about 6 million miles per hour. The interaction of the UV radiation and the fast wind with the ejected red giant envelope creates the planetary nebula, shown by the large spherical shell in the bigger image.
In rare cases, nuclear fusion reactions in the region surrounding the star's core heat the outer envelope of the star so much that it temporarily becomes a red giant again. The sequence of events - envelope ejection followed by a fast stellar wind - is repeated on a much faster scale than before, and a small-scale planetary nebula is created inside the original one. In a sense, the planetary nebula is reborn.
The large nebula seen in the larger image has an observed age of about 12,500 years and was formed by the initial interaction of the fast and slow winds. The cloverleaf pattern of knots seen in both images, correspond to the recently ejected material. These knots were produced much more recently, as they have an observed age of about 850 years, based on observations of their expansion using HST.
The diffuse X-ray emission seen in the larger image and in the region around the central source in the inset is caused by interactions between wind from the star and the knots of the ejected material. The knots are heated and eroded by this interaction, producing X-ray emission. The cause of the point-like X-ray emission from the central star is unknown.
Studies of A30 and other planetary nebulas help improve our understanding of the evolution of sun-like stars as they near the end of their lifetime. The X-ray emission reveals how the material lost by the stars at different evolutionary stages interact with each another. These observations of A30, located about 5500 light years away, provide a picture of the harsh environment that the solar system will evolve towards in several billion years, when the sun's strong stellar wind and energetic radiation will blast those planets that survived the previous, red giant phase of stellar evolution.
The structures seen in A30 originally inspired the idea of reborn planetary nebulas, and only three other examples of this phenomenon are known. A new study of A30, using the observatories mentioned above, has been reported by an international team of astronomers in the August 20th, 2012 issue of The Astrophysical Journal.
###
The first author of the paper reporting these results is Martn A. Guerrero of the Instituto de Astrofsica de Andaluca (IAA-CSIC) in Spain. The other authors are N. Ruiz, also from the IAA-CSIC, Spain; W.-R. Hamann, from the University of Potsdam, Germany; Y.-H. Chu, from the University of Illinois, Urbana, IL; H. Todt, from the University of Potsdam, Germany; D. Schnberner, from the Leibniz-Institut Fr Astrophysik in Potsdam, Germany; L. Oskinova, from the University of Potsdam, Germany; R. Gruendl, from the University of Illinois, Urbana, IL; M. Steffen, from the Leibniz-Institut Fr Astrophysik in Potsdam, Germany; W. Blair, from Johns Hopkins University in Baltimore, MD and J. Toal from the IAA-CSIC, Spain.
NASA's Marshall Space Flight Center in Huntsville, Ala., manages the Chandra program for NASA's Science Mission Directorate in Washington. The Smithsonian Astrophysical Observatory controls Chandra's science and flight operations from Cambridge, Mass.
[ | E-mail | Share ] ?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Source: http://www.eurekalert.org/pub_releases/2012-11/cxc-xfa111512.php
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MISSOULA ?
The University of Montana will interview four candidates for the position of vice president for integrated communications in coming weeks.
The candidates are Scott Ochander, Valparaiso University vice president for integrated marketing and communications-greater Chicago area; Mario Schulzke, founder of IdeaMensch, a digital publication aimed at helping entrepreneurs; Kari Dilloo, group communications manager with Microsoft/Bing; and Libby Eckhardt, chief communications officer for the College of Arts and Sciences at Ohio State University.
The vice president for integrated communications is the chief communications officer for the University and will provide leadership for UM marketing and branding, licensing, social media and all other communications. This position directly oversees the Broadcast Media Center, Printing and Graphic Services, and University Relations.
?After an extensive national search, we have pinpointed four exciting candidates with the potential to enhance our University?s communications strategies and take our marketing programs to a higher level,? said Peggy Kuhr, interim vice president for integrated communications and chairwoman of the 16-member search committee. ?We invite campus and community members to attend the candidate forums and help select the right person for this vital leadership position.?
Public forums are scheduled for:
Ochander has served as the chief marketing officer of Indiana?s Valparaiso University since 2009. Previous positions include senior vice president of market innovation for New Edge + The Brewery in Seattle and London, senior director of marketing for the Pacific Northwest National Laboratory in Richland, Wash., and senior marketing and brand leader for multiple Dow Chemical Co. units. He received an MBA in marketing from Central Michigan University and has bachelor?s degrees in business administration and fine arts from Western Michigan University.
Schulzke founded his IdeaMensch digital publication (http://ideamensch.com) in 2009 and at the same time was senior director of digital strategy for WDCW (Wong, Doody, Crandall, Wiener) in Los Angeles. Previously, he was director of new media for R2C Group in Portland, Ore., and an instructor at the Academy of Art University in San Francisco. He also oversaw business development and public relations for WDCW. He received a Bachelor of Science degree in business administration with a minor in Spanish from UM.
Dilloo has been employed by Microsoft since 2006, working as group public relations manager for the Xbox 360, group communications manager for the Entertainment and Devices Division, and group communications manager for Microsoft/Bing. Before joining Microsoft, she held a number of positions in the Seattle area, including consumer public relations manager with Expedia Inc., senior account executive with Edelman and senior account executive with Imagio/JWT. She received a Bachelor of Science degree in business administration with an emphasis in marketing and a minor in communications from UM.
Eckhardt has worked at Ohio State since 2007, serving first as director of marketing and communications for Facilities Operations and Development and then as chief communications officer for the College of Arts and Sciences. Before joining OSU, she worked as an account executive and project manager for Lorz Communications Inc. in Columbus, Ohio, and as a media coordinator for the Music Maker Relief Foundation in Hillsborough, N.C. Eckhardt received a Bachelor of Arts degree in journalism from OSU and an Associate of Arts degree from Columbus State University.
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CBS
Western Montana, Dailies
111312vpic
Source: http://news.umt.edu/2012/11/111312vpic.aspx
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?How do you really know if your business is growing if you don?t track sales data? Forecast sales, track deals, and manage client relationships with the help of a free simple CRM and sales app like Base.
?
Some businesses grow too fast and can?t sustain it.? Some businesses sacrifice customer satisfaction for revenue.? Be careful or your growth desires could put you out of business.
Growth is something most businesses strive for. Complacency is not a good thing, but growth needs to be carefully planned for it to be sustainable.
Companies that try to grow too fast may have problems ? problems with keeping their customers as well as problems succeeding at what they are trying to accomplish.? Don?t forfeit your current success in the name of growth. Careful planning can help you grow your business, sell more, attract new customers, and make more money. But if you aren?t strategic in your growth, you can lose the success you have today and jeopardize future sustainability.
You need to find a way to grow your small business while ensuring that you can properly service the business that you already have. The wrong approach could be a catastrophe.
The right infrastructure needs to be in place to help you grow.
You will need people, processes, and you may need a reserve of funds, too. Funds could be for marketing, for inventory, technology, and so on. Beyond these things, you?ll need relationships in place so that you can serve new business that comes in.
You need the right infrastructure for new and existing business.
Always operate with a customer experience mindset. If you put the customer first, you?ll do great ?A good experience dealing with you will lead to repeat business and referrals and customer loyalty.
Do you know what your market wants?
Know what makes your customers happy and this can help you achieve sustainable growth. Never stop researching so that you can continue to sustain your success by knowing what your customers want.
Tracking is paramount to success with growth and to managing your customer relationships.? Make sure you track existing deals, new deals, potential leads, and customer details. When companies experience a large influx in volume, things can fall between the cracks.? A spreadsheet won?t cut it. A spreadsheet can?t forecast for your small business.
Good tracking tools can save you from having to micromanage staff, too, and can open time up in your calendar so that you can confidently lead the growth of your business. Tracking can lead to customer satisfaction because you?ll know by your tracking efforts just what to do to keep customers satisfied.
As you strive to grow you need to analyze your progress. The data you gather can help you see where things are going well and where things may need tweaks.
Companies who experience growth need to sow seeds for future expansion. ?Invest in tools and people so that you can expand on your current success and keep good things coming.
Growth is great. However, not everyone can achieve it. Even fewer can take make the jump to the next level rather than just seeing a temporary spike in volume.? Make customer satisfaction, for old and new customers, your priority and you?ll be in a great position to succeed.
?
Dana Prince writes for Future Simple?s Growth University. Dana blends her passion for entrepreneurship with experience in software licensing sales, product management, online marketing, and SEO writing to run Dana Prince Writing, a web writing agency that creates optimized content that helps websites succeed.Source: http://www.futuresimple.com/blog/growth-small-business-success/
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Source: http://www.blackspreen.org/local-business-internet-marketing-course-review.html
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